The pathophysiology of Traumatic Optic Neuropathy (TON) include a primary and secondary
mechanism of injury. At present, no studies validate a particular approach to the management
of TON. There are three management lines for these patients that include 1)observation
only;2)medical treatment with high or megadoses of methylprednisolone; and 3)surgical
intervention. Studies have shown that forces applied to the frontal bone and malar eminences
are transferred and concentrated in the area near the optic canal. The tight adherence of the
optic nerve's dural sheath to the periosteum within the optic canal is also thought to
contribute to this segment of the nerve being extremely susceptible to the deformative
stresses of the skull bones. In this study, investigators aim to make a randomized controlled
trial to certify the efficiency of optic nerve canal decompression for TON patients.