Recent human studies found tissue sodium storage in patients with hyperaldosteronism that
could be detected non-invasively by 23Na-MRI. Tissue sodium accumulation could be mobilized
upon treatment of hyperaldosteronism. Besides, former animal studies applying chemical
electrolyte analysis indicate that this aldosterone induced sodium storage might be
accompanied by intracellular potassium loss. Wether such an intracellular tissue Potassium
loss occurs in vivo in patients with hyperaldosteronism and if this deficiency can be
corrected by treatment is unclear. The investigators will employ 39K-MR Imaging at 7Tesla to
further assess this hypothesis.